A new report from researchers at Japan’s Riken Research Institute predicts the world is set for a post-COVID-19 era heart failure pandemic thanks to the impact of catching and suffering from SARS-CoV-2. Published in Cell on December 22, 2023, the paper asserts that patients with chronic cardiomyopathy may have persistent viral infections in their hearts, particularly with SARS-CoV-2, which they explain targets the ACE2 receptor highly expressed in human hearts. With no mention of the deadly mRNA COVID-19 gene-damaging jabs, the authors insist this situation introduces concerns about a potential global heart failure pandemic stemming from the COVID-19 SARS-CoV-2 infection in the near future.
Are we really four years into the most unprecedented health catastrophe of our lifetime, and yet specialists studying the ramifications of the disaster are okay with omitting the most crucial element of the entire debacle? Of course, that critical component is the experimental mRNA jabs that have been under development for decades by the Department of Defense (DARPA) and were carelessly tested on a global scale during the COVID-19 pandemic. Make no mistake; the rapidly produced mRNA shots provide a much-needed large-scale clinical trial to help steer the hugely lucrative mRNA platform, with those jabbed serving as the clueless guinea pigs.
Emphasizing the need for “countermeasures,” the paper, titled “Predicted risk of heart failure pandemic due to persistent SARS-CoV-2 infection using a three-dimensional cardiac model,” reports that coronavirus infection occurs when a protruding spike protein on the virus’s surface latches onto ACE2 receptors on the surface of human cells. According to the team, the ACE2 receptor is more common in the heart than in other organs. They state that some COVID patients have reportedly had reduced cardiac function, “but the mechanism’s details are not yet known.” Hmmm, could it be the dangerous COVID-19 “vaccine.” Many experts, including esteemed cardiologist Dr. Peter McCullough think so. Apparently, the paper researchers don’t consider the jab to be a possibility.
The only mention of anything related to vaccines is the word vaccination, which occurs once. Explaining we must move past focusing on “acute phase symptoms” and on to “chronic health problems,” the authors mention vaccination in their introduction, stating, “The dominant SARS-CoV-2 mutant strain continues to be the BA.5 strain (omicron strain) worldwide. Case fatality risk and hospitalization is reduced in the delta and omicron strains compared to earlier strains due to several factors, such as a reduction in the inherent severity of the virus, an increase in spontaneous outbreaks, vaccination, and the use of therapeutic agents.” That’s it. Stating that future heart failure due to persistent SARS-CoV-2 infection is expected to increase exponentially, they go on to add:
“Even though conclusive clinical evidence that persistent SARS-CoV-2 infection is associated with declined cardiac function has not been reported so far, the proof-of-concept study of the possibility of SARS-CoV-2 persistent infection of the heart and the potential risk of opportunistic progression of heart failure should be validated by a three-dimensional human cardiac tissue model which would serve as the alarm bell for a global healthcare risk.”
Interestingly, one of the study authors, Riken Research Leader Hidetoshi Masumoto, was a discussion leader at the Evening Session of the 15th U.S. Japanese Symposium on Drug Delivery Systems that took place in Hawaii on December 14, 2019. His session chair was Dan Wattendorf of the Bill and Melinda Gates Foundation. Considering Riken’s heavy hand in growing Artificial Intelligence (AI)—and understanding they were a crucial part of addressing the pandemic—any connection to Bill Gates in studies that leave out vital components (like the mRNA vaccine as a potential contributor to heart problems) is instantly suspicious.
Moving on past Bill Gates, for their research—that ultimately concluded a heart failure pandemic is on the horizon—the team first created heart tissue using induced pluripotent stem (iPS) cells. When a large amount of the SARS-CoV-2 virus was made to infect the tissue, the researchers reported that cardiac function declined and did not recover. They explain that when 10 percent of the previous amount infected the tissue, a certain level of cardiac function remained, but the infection persisted for four weeks. The researchers remarked it is possible that some patients won’t develop heart failure even if the infection persists.
Furthermore, the study declared that when cardiac tissue was placed under hypoxic conditions to reduce cardiac function, uninfected cells recovered after a specific time, but cells that remained infected with a small amount of coronavirus did not recover. The researchers noted that their recovery ability was weakened by persistent infection.
For those unaware, induced pluripotent stem (iPS) cells are a type of pluripotent stem cell derived from adult somatic cells that have been genetically reprogrammed to an embryonic stem (ES) cell-like state through the forced expression of genes and factors essential for maintaining the defining properties of ES cells. The lab-created iPS cells can be used to assemble cells that replicate tissues affected by disease. This technique offers the possibility of using iPS cells to test drug efficacy, side effects, and toxicity and to develop new drugs and therapies.
From all accounts, the research paper’s conclusions no doubt support their mission to develop a three-dimensional human cardiac tissue model that would serve as the alarm bell for a global healthcare risk. Their investigation using the human iPS cell-based cardiac tissue was the first research to experimentally demonstrate that SARS-CoV-2 persistent infection of the human heart is at high risk of cardiac dysfunction with additional hypoxic stress. Undoubtedly hoping to expand their in vitro, AI-reliant research—which they admit does not fully represent the diverse factors existing within the human body—the researchers wrote:
“The development of a multi-organ model, such as MultiCUBE platform, which reproduces the biological processes that occur in the human body as organ-on-a-chip would greatly aid in the investigation of interactions between different organs and potentially help unravel the possible route of SARS-CoV-2 infection to the heart in the future.
In conclusion, this report may serve as a warning for the possibility of a heart failure pandemic in the post COVID-19 era. As a countermeasure against this global healthcare risk, this model would serve as a useful tool to investigate the mechanism of the onset and the progression of SARS-CoV-2 cardiomyopathy and to develop therapeutic options.”
Instead of stoking fears by predicting a heart failure pandemic resulting from SARS-CoV-2 infection, the time is now for researchers to pull the COVID-19 “vaccine” tyranny doors wide open and investigate the much more extensive heart damage—often resulting in death—that absolutely exists above and beyond the infection itself. Tellingly, the mRNA COVID jab was mass-distributed under forced conditions despite full knowledge of its destruction. Undoubtedly linked to bioterrorism, the controlling motive behind the jabs—to merge humans and technology into one—is poised to impact not just the health of humanity but our very existence as freedom-loving, sentient human beings.
There is overwhelming evidence that Covid was a bioweapon that escaped from a lab in Wuhan, China. I detail the case in my new book, “The Wuhan Cover-Up.” pic.twitter.com/RMUFzMcxvD
— Robert F. Kennedy Jr (@RobertKennedyJr) December 28, 2023