A recent peer-reviewed study has emerged with findings that quietly confirm that the dreaded and debilitating Long COVID is the result of the experimental COVID-19 injections, not the virus itself, as many would like us to believe. Tellingly, every single vaccinated participant in the study was found to have fibrinolysis-resistant, Thioflavin-T-positive amyloid microclots circulating in their blood. Yes, you read that correctly: 100% of the participants were vaccinated. And yet, the authors frame these findings in the familiar language of “Long COVID” from the virus, a narrative choice that, upon closer inspection, reveals that Long COVID occurred almost entirely in those heavily vaccinated, without any lab confirmation of previously being infected with SARS-CoV-2. Thus, as pointed out by Nicolas Hulscher, MPH:

“In reality, the study is observing Long VACCINE pathology, not Long COVID.” 

To be clear, the data underpinning the paper are not in dispute. It is the origin of Long COVID that presents the problem. The use of Thioflavin-T—a dye that binds specifically to amyloid structures—means that what the researchers observed were, by definition, amyloid microclots. And they were everywhere, meaning small ones in everyone, but particularly large, pathogenic ones in those labeled as having “Long COVID.” Or, perhaps more accurately, in those suffering from Long COVID resulting from what should more appropriately be named gene-damaging mRNA shot syndrome.

It is at this point in the study, it becomes hard to ignore the destruction of the toxic COVID-19 injections. Of the 88 participants, 83 were vaccinated. The researchers claimed to study post-infection sequelae, yet conducted no PCR tests, no antibody testing, and no viral sequencing. Why is this oversight critical? Because it means they performed no testing to confirm that any of the study participants had ever had COVID-19. Instead, a diagnosis was made based on symptoms and a clinical hunch. It’s as if a weather report declared a hurricane based solely on someone showing up with wet socks.

More damningly, the study’s own in vitro experiments demonstrated that purified spike protein alone was enough to produce identical, misfolded fibrin structures. That is not just a pattern. Instead, it is causation in uniform, saluting the data. Still, in what can only be described as scientific auto-censorship, the authors avoid the most obvious conclusion, which is that the pathology they observed may not be the result of the virus, but instead the result of its simulated evil twin—the spike protein introduced by mRNA and adenoviral-vector vaccines. The distinction is not trivial. If compared to a fire, it is the difference between a forest fire and a controlled burn gone rogue.

The data paint a vivid picture, showing that every vaccinated participant, including so-called healthy controls, had amyloid microclots in their bloodstream. Not some. Not most. Every single one of them. And when broken down by symptom severity, the picture grows even shadier. Nearly all those labeled with “Long COVID” had large amyloid clots, and over half had very large ones—those in the >1600 µm² range. And this wasn’t an isolated anomaly. The severity of symptoms marched in lockstep with the intensity of vascular damage.

And yet, again, no participant was confirmed to have had COVID-19. While not surprising, given the manipulated crisis that is/was the COVID-19 pandemic, that omission alone is staggering. Without evidence of SARS-CoV-2 infection, what exactly are they going to great lengths to label as “Long COVID”? Significantly, the study’s authors inadvertently demonstrated that these clotting anomalies are not dependent on SARS-CoV-2 infection at all. Again, they seem far more plausibly linked to exposure to the spike protein itself.

But it gets worse. Even more striking is that the study directly added the spike protein to fibrinogen in vitro. The result? Identical misfolded fibrin clots—amyloidogenic, fibrinolysis-resistant, and bound by Thioflavin-T. This smoking gun opens a wormhole of uncomfortable questions. If microclots are present in 100% of vaccinated individuals, are we witnessing a slow-motion vascular time bomb? What happens when billions of people accumulate microscopic, insoluble clots over years? Unfortunately, the study doesn’t answer these questions. Nonetheless, it dares us to ask them.

Of course, it’s easy—and often correct—to be skeptical of sensationalism in science. Why? Because we are often told that extraordinary claims require extraordinary evidence. Fair enough. But this study, while imperfect, is in no way extraordinary in its design. It is a straightforward study. In fact, it feels like the scientific equivalent of a smoke detector. No five-alarm fire, just a quiet signal that something is on fire. And what’s smoldering might be far more than a few capillaries. What’s smoldering is the narrative integrity of the entire pandemic response – a response that aimed to alter life as we know it in the most tyrannical ways.

Of course, in any endeavor, it’s essential not to overreach. This paper does not claim that vaccination is universally harmful, nor does it track long-term health outcomes. It doesn’t follow participants over time to see who develops cardiovascular disease, stroke, or other clot-related issues. But what it does do—whether intentionally or not—is provide biochemical fingerprints that mirror postmortem findings reported by embalmers, forensic analysts, and a growing number of clinicians around the world. This cannot be overlooked.

For example, as noted by Hulscher, at the recent Tennessee Funeral Directors Association convention, 64% of embalmers reported encountering unusual, white fibrous clots in corpses—structures found in 17% of all bodies. Before 2021, this condition was almost never observed. Forensic analysis has identified these white fibrous clots as amyloidogenic fibrin masses, dense and rubbery, resistant to decay. Sound familiar?

Clearly, this eye-opening study is deeply suggestive. Science, after all, is not about certainty—it’s about curiosity disciplined by method. And when a method reveals something unsettling, it is not anti-scientific to explore it. Instead, it is anti-scientific to ignore it. Of course, it is critical to understand the conflicts of interest entangling the authors of the study. Specifically, as related to their Diagnostic Method for Long COVID PCT patent application and their involvement in Biocode Technologies (a Bill Gates-funded Stellenbosch University start-up company), whose Long-COVID-focused website boldly states:

“Our patented diagnostic technology detects abnormal microclots in the blood, indicating poor vascular health, inflammation, and an increased risk for thrombotic endothelialitis. This pioneering test has been used to detect and aid in the treatment of microclots in thousands with Long COVID, a debilitating condition impacting millions worldwide and deeply affecting global health, society, and the economy.”

So here we are, talking about a study that politely avoids its own implications for the sake of a hefty profit, no doubt. Which begs the question: Do we really have a scientific community too lured by greed and too spooked by controversy to interpret its own data plainly and openly? The blatant oversight by suggestive studies such as this leaves the scarred public, still reeling from the information warfare of recent years, left to piece together their own analysis based on half-truths and silenced cues.

Despite this, the message is clear. If the spike protein can induce amyloid microclots, and if those clots are found in every vaccinated person examined, then the conversation we need to have is not only about turbo cancers, myocarditis in teenagers, or sudden collapses in athletes, among other disasters. It is much bigger and all-encompassing. For Long COVID, it is about the fundamental vascular integrity of the human population in the aftermath of a mass pharmacological experiment coercively injected into the arms of society. That’s not a conspiracy theory.

As Long COVID persists—and the dangerous COVID-19 shots remain on the market—the findings of this study deserve the full attention of medicine, media, and government alike. To shrug off the bombshell details buried in this study and instead carry on insisting that the SARS-CoV-2 virus is the cause of Long COVID would be yet another egregious crime against humanity.

 

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Tracy Beanz & Michelle Edwards

Tracy Beanz is an investigative journalist, Editor-in-Chief of UncoverDC, and host of the daily With Beanz podcast. She gained recognition for her in-depth coverage of the COVID-19 crisis, breaking major stories on the virus’s origin, timeline, and the bureaucratic corruption surrounding early treatment and the mRNA vaccine rollout. Tracy is also widely known for reporting on Murthy v. Missouri (Formerly Missouri v. Biden), a landmark free speech case challenging government-imposed censorship of doctors and others who presented alternative viewpoints during the pandemic.